Matrix sDtiffness induces epithelial–mesenchymal transition and promotes chemoresistance in pancreatic cancer cells
Matrix sDtiffness induces epithelial–mesenchymal transition and promotes chemoresistance in pancreatic cancer cells
ATRA mechanically reprograms pancreatic stellate cells to suppress matrix remodelling and inhibit cancer cell invasion
ATRA mechanically reprograms pancreatic stellate cells to suppress matrix remodelling and inhibit cancer cell invasion
Matrix stiffness modulates the activity of MMP-9 and TIMP-1 in hepatic stellate cells to perpetuate fibrosis
Matrix stiffness modulates the activity of MMP-9 and TIMP-1 in hepatic stellate cells to perpetuate fibrosis
Substrate Rigidity Controls Activation and Durotaxis in Pancreatic Stellate Cells
Substrate Rigidity Controls Activation and Durotaxis in Pancreatic Stellate Cells
Syndecan-4 tunes cell mechanics by activating the kindlin-integrin-RhoA pathway
Tamoxifen mechanically reprograms the tumor microenvironment via HIF‐1A and reduces cancer cell survival
Tamoxifen mechanically reprograms the tumor microenvironment via HIF‐1A and reduces cancer cell survival
ATRA modulates mechanical activation of TGF-β by pancreatic stellate cells
ATRA modulates mechanical activation of TGF-β by pancreatic stellate cells
Adhesive ligand tether length affects the size and length of focal adhesions and influences cell spreading and attachment
Adhesive ligand tether length affects the size and length of focal adhesions and influences cell spreading and attachment
GPER is a mechanoregulator of pancreatic stellate cells and the tumor microenvironment
GPER is a mechanoregulator of pancreatic stellate cells and the tumor microenvironment
FAK controls the mechanical activation of YAP, a transcriptional regulator required for durotaxis